In its mildest form, minor changes occur in the choroid, which is the vascular layer in the back of the eye. These changes have little effect on sight. CEA affects the retina, the choroid, and in severe cases, the sclera and optic nerve.
CEA is an inherited disease and most of the eye lesions associated with CEA are present at birth. But minor lesions may not be detectable on the eye examination after three months of age, so collies intended for breeding programs should be examined early at six to seven weeks of age. Even dogs with minor lesions should not be bred because their offspring may be affected with more serious forms of the disorder.
CEA is a simple recessive disorder. All affected animals have two genes for CEA; therefore, both of their parents are either affected dogs or carriers.
What to Watch For
Most dogs with CEA have no detectable changes in vision; however, if they have large lesions, they may have visual defects.
Diagnosis
CEA is best diagnosed at between six and 12 weeks of age. CEA is diagnosed based upon the finding of any of the eye lesions described above.
The primary tool used to diagnose CEA is indirect funduscopy. This test examines the retina or fundus of the eye, which is the back portion of the interior of the eyeball. It involves examining the retina through the pupil using a lens and ophthalmoscope.
Treatment
Currently, there is no treatment for CEA. The major emphasis is placed on eliminating it from affected breeds.
Home Care and Prevention
Dogs with only choroidal hypoplasia or small colobomas do not need follow-up care. Dogs with retinal hemorrhages and large colobomas may be re-examined in two to three months to monitor for the development of retinal detachments.
The disease can best be prevented by breeding only normal dogs.
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